RT Journal Article
SR Electronic
T1 The Identity and Parentage of the Variety Known in California as Petite Sirah
JF American Journal of Enology and Viticulture
JO Am J Enol Vitic.
FD American Society for Enology and Viticulture
SP 236
OP 242
DO 10.5344/ajev.1999.50.3.236
VO 50
IS 3
A1 Carole P. Meredith
A1 John E. Bowers
A1 Summaira Riaz
A1 Vanessa Handley
A1 Elizabeth B. Bandman
A1 Gerald S. Dangl
YR 1999
UL http://www.ajevonline.org/content/50/3/236.abstract
AB DNA marker analysis was used to determine the varietal identity of Petite Sirah in public collections and commercial vineyards in California. Twenty-one vines analyzed from public collections at the University of California at Davis included accessions labeled Petite Sirah, Durif, Syrah and Serine. Fifty-three vines from 26 private Petite Sirah vineyards in four California counties were also analyzed. Several accessions each of Durif, Peloursin, and Syrah obtained from Montpellier, France and an accession of Pinot noir from the University of California at Davis were used as controls for varietal identification. Samples were analyzed with four to eight simple sequence repeat (SSR) DNA markers. Some samples were first analyzed with four DNA probes to detect restriction fragment length polymorphisms (RFLPs). Davis accessions labeled Petite Sirah were found to include vines that we determined to be Durif, Peloursin, Syrah, and Pinot noir. Accessions labeled Durif' included vines identical to Durif and Peloursin. The Syrah accessions were identical to the Syrah controls. The Serine accession was found to be Pinot noir. Forty-nine of the 53 Petite Sirah vines from private vineyards were identical to Durif. Four vines, from three vineyards in two counties, were Peloursin. Comparison of the SSR genotypes of Durif and Peloursin indicates that Durif is probably a seedling of Peloursin as reported and cannot be a selection of Peloursin (as also reported). The other parent of Durif is most probably Syrah. SSR genotypes of Durif, Peloursin, and Syrah at 25 loci are consistent with this relationship and likelihood analysis of SSR allele frequencies supports the relationship with a very high degree of probability.